Monsonego further.

.. Monsonego further. Like in other mouse models of the disease, it is shown that with aging a-beta aggregates accumulate in brain areas of cognitive functions and stimulate an inflammatory response in the brain, however, stimulation of an immune response to A. Dr. Mice not only a highly efficient clearance of A-beta from the brain results, but in a significantly reduced inflammatory response. In addition, we were able to predict that the characteristics of the immune response in mice were the same as in humans. This study thus provides the basis for the development of an individual-based immunotherapeutic approach to Alzheimer since different populations vary based on a vaccine to their genetic background Monsonego explained. As we proved we can expect specific answers for several rich genes in the population, further studies are necessary safety and efficacy safety and efficacy in our humanized mouse model of AD.

Altmann, Howard L. Weiner* and Alon Monsonego3,* Center for Neurologic Diseases, Harvard Medical School and Brigham and Women’s Hospital, MA, the Shraga Segal Department of Microbiology and Immunology, Faculty of Health Sciences, and the National Institute for Biotechnology in the Negev, Ben-Gurion University of the Negev, Beer – Sheva, Israel and Human Disease Immunogenetics Group, Department of Infectious Diseases and Transplantation Biology Group, Medical Research Council Clinical Sciences Centre, Imperial College School of Medicine, Hammersmith Hospital, United Kingdom..This area of research of the European Commission by the European Commission for several years. In fact, the project awarded 12 of to the EC Programme following also of Christine Petit out which accommodated order the prominent role of transmission in isolated , congenital or early-onset forms of deafness boyhood came to light.

Hear euros broadens the scope which investigation to the elderly include: presbycusis , a inner ear hearing loss form of deafness, for majority of cases of hearing loss occurs at human 40 years of age. The hypothesis that these age-related hearing has a genetic basis was raised decades of and share. Only recently the scientific has proven that 50 percent of the loss can be attributed to genetic factors. Age deafness is a complex trait influences by the interaction between environmental and genetic risk factors.